Which of the following is not a function of gastric hydrochloric acid (HCI)?
Destroy ingested pathogens
Convert Fe3+ to Fe2+
Emulsify lipids
Activate pepsinogen to pepsin
The Correct Answer is C
A. Destroy ingested pathogens: HCl in the stomach helps destroy ingested pathogens, providing a first line of defense against infections. Therefore, this is a correct function of HCl.
B. Convert Fe3+ to Fe2+: HCl aids in the conversion of iron from its ferric (Fe3+) to ferrous (Fe2+) form, which is more easily absorbed in the intestine. Therefore, this is a correct function of HCl.
C. Emulsify lipids: Emulsification of lipids is the function of bile, not hydrochloric acid. HCl does not emulsify lipids. This is the correct answer.
D. Activate pepsinogen to pepsin: HCl activates pepsinogen, converting it to the active enzyme pepsin, which is essential for protein digestion in the stomach. Therefore, this is a correct function of HCl.
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Correct Answer is A
Explanation
A. High HDL: low LDL- High-density lipoprotein (HDL) is known as "good cholesterol" because it helps remove cholesterol from the arteries. Low-density lipoprotein (LDL) is known as "bad cholesterol" because it can lead to plaque buildup in arteries. A high HDL to low LDL ratio is considered healthy as it is associated with a lower risk of cardiovascular disease.
B. High LDL: low chylomicron- High levels of LDL are unhealthy as they increase the risk of plaque formation in arteries, which can lead to cardiovascular diseases. Chylomicrons are lipoproteins that transport triglycerides and cholesterol from the intestines to other parts of the body.
C. High SFA: low HDL- Saturated fatty acids (SFA) can raise LDL levels and lower HDL levels, leading to an unhealthy lipid profile and increased risk of heart disease.
D. High LDL: low HDL- High LDL and low HDL is an unhealthy combination because it increases the risk of cardiovascular disease due to higher potential for plaque buildup in the arteries.
Correct Answer is C
Explanation
A. Lactateis produced during anaerobic metabolism (when oxygen is scarce) as a byproduct of glycolysis, but it is not the direct product of glycolysis itself.
B. Carbon dioxideis a byproduct of cellular respiration, specifically during the Krebs cycle, not glycolysis.
C. Pyruvateis the end product of glycolysis, where glucose is broken down into two molecules of pyruvate.
D. Acetyl-CoAis formed from pyruvate during the transition step before the Krebs cycle, not directly from glycolysis.