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What is the significance of elevated serum levels of troponin?

A.

This intracellular substance can leak into the blood with cardiac cellular injury or death.

B.

Elevated serum levels of troponin occur because of sustained glycogenolysis.

C.

This substance is elevated when a patient has acute tubular necrosis.

D.

Serum elevation of this acute phase reactant occurs because of tissue injury.

Answer and Explanation

The Correct Answer is A

Choice A rationale

 

Elevated serum levels of troponin indicate cardiac cellular injury or death. Troponin is a protein released into the bloodstream when heart muscle cells are damaged, such as during a myocardial infarction.

 

Choice B rationale

 

Sustained glycogenolysis does not cause elevated troponin levels. Glycogenolysis is the breakdown of glycogen to glucose, primarily occurring in the liver and muscles, not related to troponin release.

 

 

Choice C rationale

 

Acute tubular necrosis does not elevate troponin levels. This condition affects the renal tubules and is typically indicated by elevated creatinine and BUN levels.

 

Choice D rationale

 

Troponin is not an acute phase reactant. Acute phase reactants are proteins whose levels change in response to inflammation, such as C-reactive protein (CRP), not troponin.


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View Related questions

Correct Answer is B

Explanation

Choice A rationale

Decreasing the secretion of thyroid stimulating hormone (TSH) by the pituitary gland is not a compensatory response to fluid volume deficit. TSH primarily regulates thyroid function and metabolism, and its secretion is not directly related to fluid balance or osmolality.

Choice B rationale

Increasing renin-angiotensin-aldosterone system (RAAS) activity by the kidneys is the body’s primary compensatory response to fluid volume deficit. When there is a decrease in blood volume or blood pressure, the kidneys release renin, which activates the RAAS. This system increases the reabsorption of sodium and water in the kidneys, leading to an increase in blood volume and blood pressure.

Choice C rationale

Decreasing the secretion of cortisol by the adrenal gland is not a compensatory response to fluid volume deficit. Cortisol is a stress hormone that helps regulate metabolism, immune response, and other functions, but it is not directly involved in fluid balance.

Choice D rationale

Increasing the secretion of natriuretic peptides by the heart is a response to fluid overload, not fluid deficit. Natriuretic peptides promote the excretion of sodium and water by the kidneys, which helps reduce blood volume and pressure. This response is opposite to what is needed in a fluid volume deficit situation.

Correct Answer is B

Explanation

Choice A rationale

Deep vein thrombosis (DVT) can lead to pulmonary embolism but does not directly cause chest pain and shortness of breath.

Choice B rationale

Pulmonary embolus (PE) is characterized by chest pain, shortness of breath, and hemoptysis (coughing up blood), especially in patients on bed rest.

Choice C rationale

Anemia can cause fatigue and shortness of breath but not typically chest pain and hemoptysis.

Choice D rationale

Right heart failure can cause shortness of breath and edema but not typically chest pain and hemoptysis.

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