Prior to chemical tests for glycosuria, clinicians checked for sweetness of the urine as a sign of
pyelitis
diabetes mellitus
renal calculus
acute glomerulonephritis
The Correct Answer is B
A. Pyelitis is an infection of the renal pelvis and does not cause sweet-smelling urine.
B. Diabetes mellitus often causes glycosuria (glucose in urine), which can make urine taste sweet.
C. Renal calculus (kidney stones) does not affect urine sweetness.
D. Acute glomerulonephritis affects kidney function and urine appearance but does not specifically cause sweet-smelling urine.
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Correct Answer is B
Explanation
A. the embryo; part of the placenta. The embryoblast (inner cell mass) will become the embryo, but the trophoblast will become part of the placenta, not the embryo.
B. part of the placenta; the embryo. The trophoblast forms part of the placenta and helps in its development, while the embryoblast (inner cell mass) will develop into the embryo.
C. part of the placenta; the yolk sac. While the trophoblast does contribute to the formation of the placenta, the embryoblast (inner cell mass) develops into the embryo, not the yolk sac. The yolk sac is derived from extraembryonic mesoderm and endoderm, not the inner cell mass directly.
D. the embryo; the yolk sac. The embryoblast (inner cell mass) develops into the embryo, but the trophoblast does not become the yolk sac. The yolk sac is formed from structures surrounding the embryo, not from the trophoblast itself.
Correct Answer is C
Explanation
A. Cutaneous transpiration: While cutaneous transpiration (evaporation of sweat from the skin) contributes to water loss, it is not the primary mechanism controlling water output.
B. Drinking: Drinking affects water intake rather than output. It does not directly control how much water is excreted from the body.
C. Urine volume: Urine volume is the primary means by which the body regulates water output. The kidneys adjust urine volume to maintain fluid balance and homeostasis.
D. Sweating: Sweating contributes to water loss but is not the primary mechanism for controlling overall water output compared to urine production.