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Prior to chemical tests for glycosuria, clinicians checked for sweetness of the urine as a sign of

A.

pyelitis

B.

diabetes mellitus

C.

renal calculus

D.

acute glomerulonephritis

Answer and Explanation

The Correct Answer is B

A. Pyelitis is an infection of the renal pelvis and does not cause sweet-smelling urine.

 

B. Diabetes mellitus often causes glycosuria (glucose in urine), which can make urine taste sweet.

 

C. Renal calculus (kidney stones) does not affect urine sweetness.

 

D. Acute glomerulonephritis affects kidney function and urine appearance but does not specifically cause sweet-smelling urine.
 


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View Related questions

Correct Answer is B

Explanation

A. Chief cells secrete pepsinogen, not HCl.

B. Parietal cells in the stomach lining are responsible for secreting hydrochloric acid (HCl). This acid is crucial for digestion and killing pathogens.

C. Mucous cells secrete mucus, which protects the stomach lining from the acidic environment. They do not secrete HCl.

D. Regenerative (stem) cells are involved in the renewal of the stomach lining, but they do not secrete HCl.

Correct Answer is B

Explanation

A. It stimulates angiotensin II secretion: Antidiuretic hormone (ADH) does not directly stimulate angiotensin II secretion; instead, it acts on the kidneys to promote water reabsorption.

B. It promotes water conservation: ADH promotes water conservation by increasing water reabsorption in the kidneys, reducing urine output, and thus helping to maintain fluid balance.

C. It inhibits salivation and thirst: ADH does not inhibit salivation or thirst; in fact, it can increase thirst to encourage fluid intake when dehydration is detected.

D. It stimulates hypothalamic osmoreceptors: While ADH affects hypothalamic osmoreceptors indirectly by promoting water retention, its primary function is to act on the kidneys to conserve water.

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