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Prior to chemical tests for glycosuria, clinicians checked for sweetness of the urine as a sign of

A.

pyelitis

B.

diabetes mellitus

C.

renal calculus

D.

acute glomerulonephritis

Answer and Explanation

The Correct Answer is B

A. Pyelitis is an infection of the renal pelvis and does not cause sweet-smelling urine.

 

B. Diabetes mellitus often causes glycosuria (glucose in urine), which can make urine taste sweet.

 

C. Renal calculus (kidney stones) does not affect urine sweetness.

 

D. Acute glomerulonephritis affects kidney function and urine appearance but does not specifically cause sweet-smelling urine.
 


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Correct Answer is D

Explanation

A. It increases both Na+ and K+ secretion: Aldosterone increases sodium (Na+) reabsorption and potassium (K+) secretion, but it does not increase the secretion of both ions.

B. It causes the urine to be more diluted. Aldosterone causes the urine to be more concentrated by increasing sodium reabsorption and potassium secretion.

C. It reduces Na+ reabsorption and K+ secretion. Aldosterone actually increases sodium reabsorption and potassium secretion, not reduces them.

D. It increases Na+ reabsorption and K+ secretion. Aldosterone promotes sodium reabsorption in the kidneys and increases potassium secretion, leading to more concentrated urine.

Correct Answer is C

Explanation

A. Lactose; lactase: Lactase is the enzyme that breaks down lactose, not the other way around.

B. Lipases; micelles: Lipases break down lipids (fats) into fatty acids and glycerol, not micelles. Micelles are formed as part of the digestion process but are not the substrate for lipases.

C. Peptidases; proteins: Peptidases (or proteases) break down proteins into smaller peptides and amino acids. This is the correct answer.

D. Lactase; glucose: Lactase breaks down lactose into glucose and galactose, not just glucose.

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