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A nurse is preparing to administer betamethasone 12mg IM to a client at 32 weeks gestation at risk for preterm delivery.
Betamethasone is available as 6mg/ml. How many ml will the nurse prepare for injection? (Use a leading zero, if it applies. Do not use a trailing zero)

A.

Do not use a trailing zero)

B.

Step 2 is: 12 รท 6 = 2 ml. Answer: 2 ml

Answer and Explanation

The Correct Answer is A

Step 1 is: 12 mg ÷ 6 mg/ml.

Step 2 is: 12 ÷ 6 = 2 ml. Answer: 2 ml


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Correct Answer is D

Explanation

Choice A rationale

A rapid pulse is not typically associated with magnesium toxicity. Magnesium toxicity more commonly affects the nervous and muscular systems.

Choice B rationale

Tingling in toes can be a sign of early magnesium sulfate effects but not necessarily toxicity. It may indicate that the medication is starting to affect the nervous system.

Choice C rationale

Cool skin temperature is not a common sign of magnesium toxicity. Symptoms of magnesium toxicity are more related to neuromuscular and respiratory function.

Choice D rationale

Absent deep tendon reflexes are a key indicator of magnesium toxicity. This finding suggests that magnesium levels are high enough to depress neuromuscular function, requiring immediate medical intervention. .

Correct Answer is C

Explanation

Choice A rationale

Increasing the rate of infusion of IV oxytocin in the presence of abnormal fetal heart rate decelerations is contraindicated. It may exacerbate uterine hyperstimulation, further compromising fetal oxygenation.

Choice B rationale

Decreasing the rate of infusion of the maintenance IV solution will not address the issue of uterine hyperstimulation or abnormal fetal heart rate decelerations. The focus should be on managing oxytocin administration.

Choice C rationale

Discontinuing the infusion of IV oxytocin is appropriate due to uterine tachysystole and associated fetal heart rate decelerations. This helps reduce uterine contractions and allows for fetal recovery, improving oxygenation.

Choice D rationale

Slowing the client's rate of breathing is not related to managing uterine contractions or fetal heart rate decelerations. The intervention should directly address the cause of the decelerations, which is oxytocin-induced hyperstimulation. .

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