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A nurse is obtaining vital signs for a client who has been taking propranolol. Which of the following findings should the nurse identify as an adverse effect of the medication?

A.

Respiratory rate 24/min

B.

Oral temperature 38.9° C (102° F)

C.

Blood pressure 118/78 mm Hg

D.

Apical pulse 50/min

Answer and Explanation

The Correct Answer is D

Rationale:

 

A. A respiratory rate of 24/min is elevated and may suggest respiratory distress, but it is not a specific adverse effect of propranolol.

 

B. An oral temperature of 38.9° C (102° F) indicates fever, which is not a typical adverse effect of propranolol.

 

C. A blood pressure of 118/78 mm Hg is within normal limits and does not indicate an adverse effect of propranolol, which is often used to manage hypertension.

 

D. An apical pulse of 50/min indicates bradycardia, a known adverse effect of propranolol, which can occur due to its action on the heart rate.


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View Related questions

Correct Answer is D

Explanation

Rationale:

A. This statement is incorrect because the client should remove contact lenses before administering brimonidine and wait at least 15 minutes before reinserting them to ensure proper absorption and avoid irritation.

B. While some mild irritation can occur, it is not a desired effect and should not be expected; the nurse should clarify what level of irritation is considered normal.

C. This statement is incorrect as brimonidine is typically a long-term treatment for glaucoma, and clients should not stop using it without consulting their provider.

D. This statement is correct; brimonidine can cause changes in eye color, particularly in individuals with lighter colored eyes, and the client should be informed about this possibility.

Correct Answer is D

Explanation

Rationale:

A. Acetylcysteine is not indicated for gastrointestinal bleeding; other treatments are used for that condition.

B. Acute bronchospasm is treated with bronchodilators rather than acetylcysteine.

C. Morphine toxicity requires other interventions, such as opioid antagonists (e.g., naloxone), and acetylcysteine is not effective in this case.

D. Acetylcysteine is specifically indicated for acetaminophen toxicity as it acts as an antidote, replenishing glutathione stores and preventing liver damage from toxic metabolites.

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