A patient with multiple sclerosis (MS) is receiving treatment with methylprednisolone during an acute exacerbation. Which of the following is the expected therapeutic effect of this treatment?
Reduction of inflammation and suppression of the immune responses
Stimulation of nerve regeneration
Improvement in cognitive function
Increase in muscle strength and coordination
The Correct Answer is A
A. Reduction of inflammation and suppression of immune responses. Methylprednisolone is a corticosteroid that reduces inflammation and suppresses immune responses, which can help manage the acute exacerbations of MS.
B. Stimulation of nerve regeneration. Methylprednisolone does not stimulate nerve regeneration; it focuses on reducing inflammation.
C. Improvement in cognitive function. Cognitive improvement is not a primary goal of methylprednisolone in MS management, though reducing inflammation may indirectly benefit cognition.
D. Increase in muscle strength and coordination. While reducing inflammation can improve symptoms, methylprednisolone itself does not directly increase muscle strength or coordination.
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Correct Answer is A
Explanation
A. Otosclerosis. Otosclerosis is a common cause of conductive hearing loss, typically due to abnormal bone growth around the stapes in the middle ear.
B. Acoustic neuroma. Acoustic neuroma is associated with sensorineural hearing loss, not conductive hearing loss.
C. Meniere's disease. Meniere's disease usually causes sensorineural hearing loss, often accompanied by vertigo and tinnitus.
D. Presbycusis. Presbycusis is age-related sensorineural hearing loss and does not result in conductive hearing loss.
Correct Answer is A
Explanation
A. Increased pulse, respirations and blood pressure with dysphagia and respiratory distress. A myasthenic crisis is characterized by severe muscle weakness that can lead to respiratory failure, dysphagia, and increased vital signs due to the stress of respiratory distress.
B. Hypotension, diarrhea, and increased salivation. These symptoms are more indicative of a cholinergic crisis, which is due to excess acetylcholine.
C. Bradycardia and hypothermia. Bradycardia and hypothermia are not characteristic signs of a myasthenic crisis.
D. Tachypnea and hyperactive deep tendon reflexes. While tachypnea can occur in respiratory distress, hyperactive reflexes are not typical in myasthenic crisis, as it involves neuromuscular weakness.